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dc.contributor.authorCaswell, Richard
dc.contributor.authorColclough, Kevin
dc.contributor.authorEllard, Sian
dc.date.accessioned2021-03-23T10:43:02Z
dc.date.available2021-03-23T10:43:02Z
dc.date.issued2020-10
dc.identifier.citationMisra S et al. Response to Comment on Misra et al. Homozygous Hypomorphic HNF1A Alleles Are a Novel Cause of Young-Onset Diabetes and Result in Sulfonylurea-Sensitive Diabetes. Diabetes Care 2020;43:909-912. Diabetes Care. 2020 Oct;43(10):e155-e156.en_US
dc.identifier.pmid32958621
dc.identifier.doi10.2337/dci20-0033
dc.identifier.urihttps://rde.dspace-express.com/handle/11287/621681
dc.language.isoenen_US
dc.publisherHighWireen_US
dc.relation.urlhttp://care.diabetesjournals.org/cgi/pmidlookup?view=long&pmid=32958621en_US
dc.rights© 2020 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. More information is available at https://www.diabetesjournals.org/content/license.en_US
dc.subjectAllelesen_US
dc.subjectDiabetes Mellitusen_US
dc.subjectHepatocyte Nuclear Factor 1-alphaen_US
dc.subjectSulfonylurea Compoundsen_US
dc.subjectWessex Classification Subject Headings::Endocrinology::Diabetesen_US
dc.titleResponse to Comment on Misra et al. Homozygous Hypomorphic HNF1A Alleles Are a Novel Cause of Young-Onset Diabetes and Result in Sulfonylurea-Sensitive Diabetes. Diabetes Care 2020;43:909-912en_US
dc.typeCommenten_US
dc.identifier.journalDiabetes Careen_US
dc.identifier.pmcidPMC7510032
dc.description.noteThis article is available to RD&E staff via NHS OpenAthens (subject to any publisher embargo). Click on the Publisher URL, and log in with NHS OpenAthens if prompted.en_US
dc.description.fundingThis work was undertaken with funds from the Diabetes Research and Wellness Foundation and the Imperial College Healthcare Charity and with infrastructure support from the National Institute for Health Research (NIHR) Imperial Biomedical Research Centre (BRC), Imperial Clin ical Research Facility, and Clinical Research Network. S.M. is currently supported by a Future Leaders Mentorship award from the European Association for the Study of Diabetes. A.J. was a Diabetes UK George Alberti Clinical Research Fellow when contributing to this study. S.E. is a Wellcome Trust Senior Investigator. A.L.G. is a Wellcome Senior Fellow in Basic Biomedical Science. This work was funded in Oxford by the Wellcome Trust (095101 and 200837 to A.L.G.). The research was also funded by the NIHR Oxford and BRC (to A.L.G.).en_US
dc.type.versionPublisheden_US
dc.description.admin-noteaccepted version, submitted versionen_US


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