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    • Diagnostic genetic testing for monogenic diabetes and congenital hyperinsulinemia 

      Houghton, Jane A. L. (Springer, 2020-01)
      Monogenic diabetes and hyperinsulinism are genetically heterogeneous disorders. The determination of the genetic etiology defines the diagnostic subtype, predicts prognosis, and importantly can guide clinical management. ...
    • Assessment of MTNR1B type 2 diabetes genetic risk modification by shift work and Morningness-Eveningness preference in the UK Biobank 

      Weedon, Michael (American Diabetes Association, 2020-02)
      Night shift work, behavioral rhythms, and the common MTNR1B risk single nucleotide polymorphism (SNP), rs10830963, associate with type 2 diabetes; however, whether they exert joint effects to exacerbate type 2 diabetes ...
    • In situ analysis reveals that CFTR is expressed in only a small minority of β-Cells in normal adult human pancreas 

      Richardson, Sarah; Morgan, Noel G (Endocrine Society, 2020-05)
      Although diabetes affects 40% to 50% of adults with cystic fibrosis, remarkably little is known regarding the underlying mechanisms leading to impaired pancreatic β-cell insulin secretion. Efforts toward improving the ...
    • Residual adrenal function in Autoimmune Addison’s Disease—effect of dual therapy with rituximab and depot tetracosactide 

      Vaidya, Bijay (Endocrine Society, 2020-04)
      In autoimmune Addison's disease (AAD), exogenous glucocorticoid (GC) therapy is an imperfect substitute for physiological GC secretion. Patients on long-term steroid replacement have increased morbidity, reduced life ...
    • Significant Benefits of AIP Testing and Clinical Screening in Familial Isolated and Young-onset Pituitary Tumors 

      Stals, Karen; Ellard, Sian (Endocrine Society, 2020-06-01)
      Context Germline mutations in the aryl hydrocarbon receptor-interacting protein (AIP) gene are responsible for a subset of familial isolated pituitary adenoma (FIPA) cases and sporadic pituitary neuroendocrine tumors ...
    • Prediction algorithms: pitfalls in interpreting genetic variants of autosomal dominant monogenic diabetes 

      Ellard, Sian; Colclough, Kevin; Patel, Kashyap A; Hattersley, Andrew T. (American Society for Clinical Investigation, 2020-01)
    • Somatostatin secretion by Na+-dependent Ca2+-induced Ca2+ release in pancreatic delta cells 

      Walker, Jonathan Neil (Springer Nature, 2020-01-20)
      Pancreatic islets are complex micro-organs consisting of at least three different cell types: glucagon-secreting alpha, insulin-producing beta and somatostatin-releasing delta cells1. Somatostatin is a powerful paracrine ...
    • Absence of islet autoantibodies and modestly raised glucose values at diabetes diagnosis should lead to testing for MODY: lessons from a 5 year pediatric Swedish national cohort study 

      Shepherd, Maggie; Ellard, Sian; Weedon, Michael; Colclough, Kevin; Hattersley, Andrew T. (American Diabetes Association, 2020-01)
      Identifying maturity-onset diabetes of the young (MODY) in pediatric populations close to diabetes diagnosis is difficult. Misdiagnosis and unnecessary insulin treatment are common. We aimed to identify the discriminatory ...
    • De Novo Mutations in EIF2B1 Affecting eIF2 Signaling Cause Neonatal/Early-Onset Diabetes and Transient Hepatic Dysfunction 

      De Franco, Elisa; Caswell, Richard; Ellard, Sian; Flanagan, Sarah E.; Hattersley, Andrew T. (American Diabetes Association, 2020-03)
      Permanent neonatal diabetes mellitus (PNDM) is caused by reduced β-cell number or impaired β-cell function. Understanding of the genetic basis of this disorder highlights fundamental β-cell mechanisms. We performed trio ...
    • Heterozygous Insulin Receptor ( INSR) Mutation associated with Neonatal Hyperinsulinemic Hypoglycaemia and Familial Diabetes Mellitus: Case Series 

      Houghton, Jayne; Colclough, Kevin; Flanagan, Sarah E. (Galenos Yayinevi, 2020-01-28)
      Mutations in the insulin receptor (INSR) gene are associated with insulin resistance and hyperglycaemia. Various autosomal dominant heterozygous INSR mutations leading to hyperinsulinemic hypoglycaemia (HH) have been ...
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    AuthorHattersley, Andrew T. (11)Colclough, Kevin (7)McDonald, Timothy J. (7)Oram, Richard A. (6)Ellard, Sian (5)Strain, W David (5)Dennis, J. M. (4)Gooding, Kim M. (3)Morgan, Noel G (3)Richardson, Sarah J. (3)... View MoreSubject
    Endocrinology (48)
    Oncology. Pathology. (8)Diabetes (4)Paediatrics (3)type 1 diabetes (3)diabetes (2)genetics (2)obesity (2)Precision Medicine (2)Type 2 Diabetes (2)... View MoreDate Issued2020 (48)Has File(s)No (26)Yes (22)

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