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    DNA Methylation Profiling in Inflammatory Bowel Disease Provides New Insights into Disease Pathogenesis.

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    URI
    http://hdl.handle.net/11287/620262
    Author
    McDermott, E.
    Ryan, E.J.
    Tosetto, M.
    Gibson, D.
    Burrage, Joe
    Keegan, D.
    Byrne, K.
    Crowe, E.
    Sexton, G.
    Malone, K.
    Harris, R.A.
    Kellermayer, R.
    Mill, Jonathan
    Cullen, G.
    Doherty, G.A.
    Mulcahy, H.
    Murphy, Therese M.
    Date
    2016-01
    Journal
    Journal of Crohn's & colitis
    Type
    Comparative Study
    Research Support, N.I.H., Extramural
    Research Support, Non-U.S. Gov't
    Publisher
    Oxford Academic
    DOI
    10.1093/ecco-jcc/jjv176
    Rights
    Archived with thanks to Journal of Crohn's & colitis
    Metadata
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    Abstract
    Inflammatory bowel diseases (IBDs) are heterogeneous disorders with complex aetiology. Quantitative genetic studies suggest that only a small proportion of the disease variance observed in IBD is accounted for by genetic variation, indicating a potential role for differential epigenetic regulation in disease aetiology. The aim of this study was to assess genome-wide DNA methylation changes specifically associated with ulcerative colitis (UC), Crohn's disease (CD) and IBD activity.
    Citation
    DNA Methylation Profiling in Inflammatory Bowel Disease Provides New Insights into Disease Pathogenesis. 2016, 10 (1):77-86 J Crohns Colitis
    Publisher URL
    https://academic.oup.com/ecco-jcc/article-lookup/doi/10.1093/ecco-jcc/jjv176
    Note
    This article is freely available via Open Access. Click on the ‘Additional Link’ above to access the full-text from the publisher’s site.
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