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dc.contributor.authorStride, A.en
dc.contributor.authorEllard, Sianen
dc.contributor.authorClark, P.en
dc.contributor.authorShakespeare, L.en
dc.contributor.authorSalzmann, Maurice B.en
dc.contributor.authorShepherd, Maggieen
dc.contributor.authorHattersley, Andrew T.en
dc.date.accessioned2016-09-02T10:29:10Z
dc.date.available2016-09-02T10:29:10Z
dc.date.issued2005-07
dc.identifier.citationBeta-cell dysfunction, insulin sensitivity, and glycosuria precede diabetes in hepatocyte nuclear factor-1alpha mutation carriers. 2005, 28 (7):1751-6 Diabetes Careen
dc.identifier.issn0149-5992
dc.identifier.pmid15983330
dc.identifier.urihttp://hdl.handle.net/11287/619275
dc.description.abstractPatients with diabetes due to hepatocyte nuclear factor (HNF)-1alpha mutations have beta-cell deficiency, insulin sensitivity, altered proinsulin levels, and a low renal threshold for glucose. It is uncertain how many of these features precede the development of diabetes. The aim of our study was to test for these characteristics in young nondiabetic HNF-1alpha mutation carriers.en
dc.description.sponsorshipWellcome Trust/United Kingdomen
dc.language.isoenen
dc.publisherHighwireen
dc.relation.urlhttp://care.diabetesjournals.org/content/28/7/1751.longen
dc.rightsArchived with thanks to Diabetes careen
dc.subjectWessex Classification Subject Headings::Oncology. Pathology.::Geneticsen
dc.titleBeta-cell dysfunction, insulin sensitivity, and glycosuria precede diabetes in hepatocyte nuclear factor-1alpha mutation carriers.en
dc.typeResearch Support, Non-U.S. Gov'ten
dc.identifier.journalDiabetes careen
dc.description.noteRD&E staff can access the full-text of this article via OpenAthens. Click on the 'Additional Link' above to access the full-text and log in with NHS OpenAthens if prompted.en
dc.type.versionPublisheden


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