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    Increased plasma incretin concentrations identifies a subset of patients with persistent congenital hyperinsulinism without KATP channel gene defects.

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    URI
    http://hdl.handle.net/11287/618199
    Author
    Shi, Y.
    Avatapalle, H. B.
    Skae, M. S.
    Padidela, R.
    Newbould, M.
    Rigby, L.
    Flanagan, Sarah
    Ellard, Sian
    Rahier, J.
    Clayton, P. E.
    Dunne, M. J.
    Banerjee, I.
    Cosgrove, K. E.
    Date
    2015-01
    Journal
    The Journal of Pediatrics
    Type
    Journal Article
    Research Support, Non-U.S. Gov't
    Publisher
    Elsevier
    DOI
    10.1016/j.jpeds.2014.09.019
    Rights
    Archived with thanks to The Journal of pediatrics
    Metadata
    Show full item record
    Abstract
    Congenital hyperinsulinism causes profound hypoglycemia, which may persist or resolve spontaneously. Among 13 children with congenital hyperinsulinism, elevated incretin hormone concentrations were detected in 2 with atypical, persistent disease. We suggest that incretin biomarkers may identify these patients, and that elevated hormone levels may contribute to their pathophysiology.
    Citation
    Increased plasma incretin concentrations identifies a subset of patients with persistent congenital hyperinsulinism without KATP channel gene defects. 2015, 166 (1):191-4 J. Pediatr.
    Publisher URL
    http://linkinghub.elsevier.com/retrieve/pii/S0022-3476(14)00847-6
    Note
    This article is freely available via Open Access, click on the 'Additional Link' above to access the full text via the publisher's site.
    Collections
    • 2015 RD&E publications
    • Honorary contracts publications
    • Molecular Genetics

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