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dc.contributor.authorOram, Richard A.en
dc.contributor.authorJones, Angus G.en
dc.contributor.authorBesser, R.en
dc.contributor.authorKnight, Bridget A.en
dc.contributor.authorShields, Beverley M.en
dc.contributor.authorBrown, Richard J.en
dc.contributor.authorHattersley, Andrew T.en
dc.contributor.authorMcDonald, Timothy J.en
dc.date.accessioned2016-08-08T11:14:23Z
dc.date.available2016-08-08T11:14:23Z
dc.date.issued2014-01
dc.identifier.citationThe majority of patients with long-duration type 1 diabetes are insulin microsecretors and have functioning beta cells. 2014, 57 (1):187-91 Diabetologiaen
dc.identifier.issn1432-0428
dc.identifier.pmid24121625
dc.identifier.doi10.1007/s00125-013-3067-x
dc.identifier.urihttp://hdl.handle.net/11287/618027
dc.description.abstractClassically, type 1 diabetes is thought to proceed to absolute insulin deficiency. Recently developed ultrasensitive assays capable of detecting C-peptide under 5 pmol/l now allow very low levels of C-peptide to be detected in patients with long-standing type 1 diabetes. It is not known whether this low-level endogenous insulin secretion responds to physiological stimuli. We aimed to assess how commonly low-level detectable C-peptide occurs in long-duration type 1 diabetes and whether it responds to a meal stimulus.en
dc.language.isoenen
dc.publisherSpringeren
dc.relation.urlhttp://dx.doi.org/10.1007/s00125-013-3067-xen
dc.rightsArchived with thanks to Diabetologiaen
dc.subjectWessex Classification Subject Headings::Endocrinology::Diabetesen
dc.titleThe majority of patients with long-duration type 1 diabetes are insulin microsecretors and have functioning beta cells.en
dc.typeJournal Articleen
dc.typeResearch Support, Non-U.S. Gov'ten
dc.identifier.journalDiabetologiaen
dc.description.noteThis article is freely available via Open Access. Click on the 'Additional Link' above to access the full-text via the publisher's site.en
dc.type.versionPublisheden


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